This coming week we want to look at each one of these HEAD/BODY IMPLANTS and remember what these several months since JAN 2019 when I became aware of the existance of these implants revealed to me as a VICTIM of illegal surveillance video and black market PORN.
First, we want to remember this----these TELEMEDICINE research and experiments are GLOBAL PRIVATE MILITARY CORPORATE research so the goals are less public health and more DEEP, DEEP REALLY DEEP STATE ----MIND-CONTROL AND SURVEILLANCE.
The goals of RETINAL IMPLANTS are to alter what our BRAINS say we see and what we REALLY see. The retina is where that visual information is sent and translated by the BRAIN. If that visual information is ALTERED at the RETINA----your BRAIN thinks it sees something different.
'The retina also contains the nerves that tell the brain what the photoreceptors are "seeing."'
The second concern about RETINAL IMPLANTS has to do with using FULL-SIGHTED PEOPLE like ME -----as CONTROLS -----not only in manipulating color and sight but in using visuals for CODING AND SIGNS tied to spying.
'Many of these are packed into the fovea, a small pit in the back of the eye that helps with the sharpness or detail of images'.
For me as a FULL-SIGHTED human having RETINAL IMPLANT illegally and without consent---installed my concern is this: These implants emit radiation and they are installed very near what is called the FOVIA where all RODS AND CONES exist. Given the numbers of electrodes connected to a RETINAL IMPLANT-----the deterioration of these RODS AND CONES would be HASTENED.
In a FULL-SIGHTED human like me----there would be no loss of color vision during a normal lifetime. Exposure to RETINAL IMPLANT EXPERIMENTATION places FULL-SIGHTED HUMANS LIKE ME----at risk of becoming that DISEASE VECTOR for vision---
PIGMENTOSIS AND MACULAR DEGENERATION.
'Factors Affecting Perceptual Threshold in Argus II Retinal ...www.ncbi.nlm.nih.gov/pmc/articles/PMC3763895When analyzing data from all subjects who have a range of light threshold levels, we found that 90.3% of electrodes placed in direct contact with the retina and within 3 mm of fovea centralis had thresholds below 1 mC/cm 2, and 80.9% had thresholds below 0.35 mC/cm 2. Therefore, while light threshold measurement can be used by clinicians and ...'
The one effect of having this RETINAL IMPLANT for about 8 years is a loss of SHARPNESS AND DETAIL in that LEFT EYE.
Below we see the factors which cause a human with HEALTHY EYES forced to have these implants slowly lose their sight:
1) An increased sensitivity to LIGHT coming through pupil. I could feel my PUPIL DILATION being controled by these IMPLANTS. These RETINAL IMPLANTS work best with MORE LIGHT coming into PUPIL ergo, they manipulated my PUPIL to open wider the normal. When this happens that increased LIGHT on my RODS AND CONES damage those RODS AND CONES over time.
2) The thermo----or heat factor from radiation emitted from ELECTRODES----also damage the RODS AND CONES over time.
Retinal safety of near infrared radiation in photovoltaic restoration of sight
H. Lorach,1,2,* J. Wang,1,2 D. Y. Lee,2,3 R. Dalal,2
P. Huie,1,2 and D. Palanker1,21Hansen Experimental Physics Laboratory, Stanford University, Stanford, CA 94305, USA 2Department of Ophthalmology, Stanford University, Stanford, CA 94305, USA 3Department of Ophthalmology, Gachon University, Gil Medical Center, Incheon, 21565, South Korea *henri.lorach@gmail.com
Abstract: Photovoltaic restoration of sight requires intense near-infrared light to effectively stimulate retinal neurons. We assess the retinal safety of such radiation with and without the retinal implant. Retinal damage threshold was determined in pigmented rabbits exposed to 880nm laser radiation. The 50% probability (ED50) of retinal damage during 100s exposures with 1.2mm diameter beam occurred at 175mW, corresponding to a modeled temperature rise of 12.5°C. With the implant, the same temperature was reached at 78mW, close to the experimental ED50 of 71mW. In typical use conditions, the retinal temperature rise is not expected to exceed 0.43°C, well within the safety limits for chronic use. ©2015 Optical Society of America
1. Introduction
Electrical stimulation of the retina allows restoration of visual perception in patients blinded by retinal degeneration
[1, 2]. Photovoltaic arrays allow a completely wireless and modular design of the implant, thereby greatly simplifying the implantation procedure [3–5]. In this approach, photodiodes convert incoming light into electric current in each pixel. Since ambient light levels are too low for generating currents sufficient for neural stimulation, more intense light has to be used. To avoid photophobia in patients with remaining light sensitivity, we utilize invisible near-infrared (NIR) wavelengths (about 880nm). To produce charge-balanced pulses of electric current, the NIR light is pulsed. Previous studies demonstrated that NIR (880nm-915nm) light elicits retinal responses to photovoltaic stimulation with irradiance ranging from 0.1 to 5mW/mm2, and pulse durations of 1 to 10ms [4, 5].
'One of the factors defining the safety limits of photovoltaic restoration of sight is the retinal heating due to light absorption, primarily in melanin (RPE and choroid) and in the silicon implant itself. The light intensity is limited by both the ocular laser safety standards (ISO 60825 and ISO 15004) and the thermal safety standards for active implantable medical devices (AIMD) (ISO 14708-1:2014 / EN 45502-1:1997). The first one defines the maximum permissible power that can enter the eye for a specific wavelength, beam size and exposure duration, whereas the second one defines a maximum temperature on the surface of an implant. Since our design involves millisecond pulses of NIR light, both safety standards are based on thermal considerations, as opposed to shorter wavelengths involving phototoxicity, or much shorter pulses (<ns), potentially involving photomechanical effects. Laser safety standards have been developed for normal eyes, but the presence of a silicon implant increases the light absorption, which will therefore decrease the maximum permissible exposure'.
Fig. 3. Retinal damage above the implants after irradiation with increasing power.
(A) Implanted retina before irradiation. (B) In the same retina, no damage can be seen at 60mW, but mild whitening is detectable at 90mW (C), pointed by the yellow arrow. (D) Severe damage and detachment occurs at 125mW.
Fig. 4. Temperature rise in the retina.
A) Spatial distribution of the temperature rise in the rabbit eye at the end of the 100s long irradiation. The temperature rise is maximum at RPE, and it rapidly drops with distance. Small absorption of the 880nm radiation in water results in slight elevation of temperature on the beam axis. B) Comparison of the thermal modeling to direct temperature measurements. Optoacoustic measurements of the RPE temperature rise during 810nm illumination by a 2mm wide beam of 129mW (dashed lines, OA1 and OA2) are plotted along the RPE temperature computed with our model using the same beam characteristics (blue curve). Experimental data replotted from (Kandulla, Elsner et al. 2006) with permission. C) Temperature rise as a function of power (dash line) plotted along with the probability of retinal damage (solid line) for the normal (red) and implanted (green) retina. At 175mW, corresponding to the ED50 in normal retina, the temperature rise reaches 12.5°C. With an implant, the same temperature is reached at 78mW (intersection between green and yellow dash lines), matching the experimental thresholds (solid green curve) with ED50 at 71mW.
________________________________________________
I estimate exposure to RETINAL IMPLANTS for about 8 years and do not seem to have lost COLOR ---but my left eye is losing SHARPNESS AND DETAIL. If this RETINAL IMPLANT is left in my LEFT EYE for another 10 years I may well have MACULAR DEGENERATION---maybe earlier.
This will have been INDUCED DELIBERATELY-------and not a NATURAL DECLINE of eyesight. Below we the normal health of MACULAR HEALTH is 90 years old. At this point as a 62 year old woman---if this RETINAL IMPLANT remains in place I will have the eyes of that 90 YEAR OLD WOMAN.
'In maculas of older adults lacking grossly visible drusen and pigmentary change (i.e., they do not have ARM), the number of cones in the cone-dominated part of the macula is stable at approximately 32,000 through the ninth decade'.
The HEALTHY humans forced and largely unaware they have these RETINAL IMPLANTS are well on the way to having HOSTING SERVER NOSY NEIGHBORS KILL THEIR EYESIGHT.
retinal degeneration, retinitis pigmentosa
Transl Vis Sci Technol. 2013 Apr; 2(4): 1.
Published online 2013 Apr 12. doi: 10.1167/tvst.2.4.1
PMCID: PMC3763895
PMID: 24049718
Factors Affecting Perceptual Threshold in Argus II Retinal Prosthesis Subjects
A. K. Ahuja,1,3 J. Yeoh,2 J. D. Dorn,1 A. Caspi,1 V. Wuyyuru,1 M. J. McMahon,1 M. S. Humayun,3 R. J. Greenberg,1 and L. daCruz2, Argus II Study
Abstract
Purpose
The Argus II epiretinal prosthesis has been developed to provide partial restoration of vision to subjects blinded from outer retinal degenerative disease. Participants were surgically implanted with the system in the United States and Europe in a single arm, prospective, multicenter clinical trial. The purpose of this investigation was to determine which factors affect electrical thresholds in order to inform surgical placement of the device.
Methods
Electrode–retina and electrode–fovea distances were determined using SD-OCT and fundus photography, respectively. Perceptual threshold to electrical stimulation of electrodes was measured using custom developed software, in which current amplitude was varied until the threshold was found. Full field stimulus light threshold was measured using the Espion D-FST test. Relationships between electrical threshold and these three explanatory variables (electrode–retina distance, electrode–fovea distance, and monocular light threshold) were quantified using regression.
Results
Regression analysis showed a significant correlation between electrical threshold and electrode–retina distance (R2 = 0.50, P = 0.0002; n = 703 electrodes). 90.3% of electrodes in contact with the macula (n = 207) elicited percepts at charge densities less than 1 mC/cm2/phase. These threshold data also correlated well with ganglion cell density profile (P = 0.03). A weaker, but still significant, inverse correlation was found between light threshold and electrical threshold (R2 < 0.52, P = 0.01). Multivariate modeling indicated that electrode–retina distance and light threshold are highly predictive of electrode threshold (R2 = 0.87; P < 0.0005).
Conclusions
Taken together, these results suggest that while light threshold should be used to inform patient selection, macular contact of the array is paramount.
Translational Relevance
Reported Argus II clinical study results are in good agreement with prior in vitro and in vivo studies, and support the development of higher-density systems that employ smaller diameter electrodes. (clinicaltrials.gov identifier: NCT00407602)
Keywords: retinal prosthesis, retinal degeneration, retinitis pigmentosa
_________________________________________
As a VICTIM of illegal, unconsented RETINAL IMPLANT experiments I KNOW these implants are on the RETINAL NERVE-----there are three positions one being right on the FOVIA-----if I have that implant RIGHT ON MY FOVIA----those RODS AND CONES will be damaged FASTER ------but, each placement does harm. Placement on the OPTIC NERVE degrades not only the RETINA but that VITAL OPTIC NERVE which is the source of AUTONOMOUS CONTROL OF EYE.
If the OPTIC NERVE deteriorate or is damaged------your eye will not automatically DILATE---it will not automatically PROCESS THE IMAGE as it normally is sent to the BRAIN.
IF THE GOAL IS TO CONTROL WHAT WE SEE-------GLOBAL BANKING 1% WOULD WANT TO KILL THAT OPTIC NERVE SO THEY COULD CREATE A VISUAL MESSAGE TO BE SENT TO THE BRAIN.
Those PARTICIPANTS already BLIND ----are seeing these MESSAGES created by global banking 1% changed before they are sent to the BRAIN. Is the ROD AND CONE and damaged eye of blind being repaired---or are they simply being SENT MESSAGES of VIRTUAL REALITY------manufactured visual sight.
Today, I still have NORMAL VISION---on the left-------while HOSTING SERVER NOSY NEIGHBORS are saying I have PIGMENTOSA. That may be true soon,.
What Is Retinitis Pigmentosa?
Articles On
What Is Retinitis? Retinitis pigmentosa (RP) is a term for a group of eye diseases that can lead to loss of sight. What they have in common is a coloring your doctor sees when he looks at your retina -- a bundle of tissue at the back of your eye. When you have RP, cells in the retina called photoreceptors don’t work the way they're supposed to, and over time, you lose your sight.
It’s a rare disorder that’s passed from parent to child. Only 1 out of every 4,000 people get it. About half of all people with RP have a family member who also has it.
The retina has two types of cells that gather light: rods and cones. The rods are around the outer ring of the retina and are active in dim light. Most forms of retinitis pigmentosa affect the rods first. Your night vision and your ability to see to the side -- peripheral vision -- go away.
Cones are mostly in the center of your retina. They help you see color and fine detail. When RP affects them, you slowly lose your central vision and your ability to see color.
Symptoms
Retinitis pigmentosa usually starts in childhood. But exactly when it starts and how quickly it gets worse varies from person to person. Most people with RP lose much of their sight by early adulthood. Then by age 40, they are often legally blind.
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Let's go back to NOSY NEIGHBORS AND THE GANG and the PSYCHO-SEXUAL TORTURE since JAN 2019. The goals of HITTING me at this point as we say was to UPGRADE these NEW BRAND IMPLANT DEVICES and BATTERIES.
The goal was to make me appear CRAZY---MENTALLY ILL ---DANGEROUS----NEEDING TO BE COMMITTED.
The COCHLEAR IMPLANT receiving FEEDBACK MESSAGES included discussion of my being AUTISTIC-----of my being SYNESTHESIA----able to see numbers and words AS COLOR. Now, I have always been CREATIVE AND A SCIENTIST who does look closely and does appreciate COLOR, TEXTURE, AND Chiaroscuro.
Never did I feel an intensity of these color sensations as was felt during PSYCHO-SEXUAL TORTURE. This is when I was saying I felt MY BODY ELECTRIC with my body feeling an AURA.
What I was feeling was a heightened stimulation of RETINAL IMPLANT directed at RODS AND CONES combined with increased RADIATION ELECTRICAL IMPULSES. The messaging coming to COCHLEAR IMPLANT makes one think you really are A AUTISTIC ASPERGER seeing in color.
All that happens inside your eye is increased stimulation of CONES ---for color ---then RODS for chiaroscuro---shades of gray. That increased intensity creates THERMAL ELECTRICAL EMISSION which makes the BRAIN feel it has an ELECTRICAL AURA.
So, while I was simply living my normal life HOSTING SERVER NOSY NEIGHBORS were saying
SHE IS AUTISTIC---THIS IS WHY SHE IS IN THIS STUDY.
This was ONE reason I was supposedly about to be sent to PSYCH WARD.
There was an increased intensity to sensations I already had.
We did not find results for:
Is synesthesia common among those with autism and Asperger syndrome? 15 Answers Zem Jones Zem Jones, works at Higher Education Answered Sep 27 2014 · Author has 845 answers and 780.4k answer views
Originally Answered:
Is synesthesia common among those with autism and Asperger's?
Almost all the people I know with synaesthesia are on the autism spectrum - I know a lot of people with synaesthesia and only a couple of them do not have an ASC diagnosis but they do have other comorbid conditions such as dyslexia.
My daughter has the strongest levels I have come across; she sees sound in colour, she "sees" people as having colours, almost like auras, her pain is described in colour (she has pain amplification - her doctors think it may be because of the way her senses cross over - and uses visualisation of the oposite colour to her pain to control it), she feels the colour of touch and texture..... Synaesthesia can cause problems and confusions, especially when people use synaesthetic language to describe things that non-synaesthetics don't experience in the same way - my daughter was learning music using a colour based system and almost fell out with her teacher, who had designed the system, because the colours weren't right. But it can be a huge boon too - many synaesthetes are highly creative in art and music, one person I know designed an intuitive data base system based on synaesthetic principles that is likely to change the way we access databases. The world for a synaesthete is a rich and vibrant place, and generally the experience of synaesthesia is very positive..
Is synesthesia common among those with autism and Asperger syndrome?
Zem Jones, works at Higher Education
Originally Answered: Is synesthesia common among those with autism and Asperger's?
Almost all the people I know with synaesthesia are on the autism spectrum - I know a lot of people with synaesthesia and only a couple of them do not have an ASC diagnosis but they do have other comorbid conditions such as dyslexia.
My daughter has the strongest levels I have come across; she sees sound in colour, she "sees" people as having colours, almost like auras, her pain is described in colour (she has pain amplification - her doctors think it may be because of the way her senses cross over - and uses visualisation of the oposite colour to her pain to control it), she feels the colour of touch and texture.....
Synaesthesia can cause problems and confusions, especially when people use synaesthetic language to describe things that non-synaesthetics don't experience in the same way - my daughter was learning music using a colour based system and almost fell out with her teacher, who had designed the system, because the colours weren't right.
But it can be a huge boon too - many synaesthetes are highly creative in art and music, one person I know designed an intuitive data base system based on synaesthetic principles that is likely to change the way we access databases. The world for a synaesthete is a rich and vibrant place, and generally the experience of synaesthesia is very positive.
____________________________
Let's go back to NOSY NEIGHBORS AND THE GANG and the PSYCHO-SEXUAL TORTURE since JAN 2019. The goals of HITTING me at this point as we say was to UPGRADE these NEW BRAND IMPLANT DEVICES and BATTERIES.
The goal was to make me appear CRAZY---MENTALLY ILL ---DANGEROUS----NEEDING TO BE COMMITTED.
The COCHLEAR IMPLANT receiving FEEDBACK MESSAGES included discussion of my being AUTISTIC-----of my being SYNESTHESIA----able to see numbers and words AS COLOR. Now, I have always been CREATIVE AND A SCIENTIST who does look closely and does appreciate COLOR, TEXTURE, AND Chiaroscuro.
Never did I feel an intensity of these color sensations as was felt during PSYCHO-SEXUAL TORTURE. This is when I was saying I felt MY BODY ELECTRIC with my body feeling an AURA.
What I was feeling was a heightened stimulation of RETINAL IMPLANT directed at RODS AND CONES combined with increased RADIATION ELECTRICAL IMPULSES. The messaging coming to COCHLEAR IMPLANT makes one think you really are A AUTISTIC ASPERGER seeing in color.
All that happens inside your eye is increased stimulation of CONES ---for color ---then RODS for chiaroscuro---shades of gray. That increased intensity creates THERMAL ELECTRICAL EMISSION which makes the BRAIN feel it has an ELECTRICAL AURA.
So, while I was simply living my normal life HOSTING SERVER NOSY NEIGHBORS were saying
SHE IS AUTISTIC---THIS IS WHY SHE IS IN THIS STUDY.
This was ONE reason I was supposedly about to be sent to PSYCH WARD.
There was an increased intensity to sensations I already had.
'youtube.com
Body Electric (LYRICS) - Lana Del Rey
*All materials used in this video are property of their respective owners.…'
I kept saying I was FEELING THE BODY ELECTRIC while these RETINAL IMPLANTS were hitting my RETINA AND OPTICAL NERVE with higher electrical impulses aimed at CONES----COLOR giving me a feeling of ELATION.
This is much like the 1960s when global banking 1% sold LSD----TAKING A TRIP as a mind-altering FAD.
PLEASE DO NOT THINK GETTING THESE RETINAL IMPLANTS WILL ENHANCE YOUR LIFE OR ABILITY TO GET A JOB----IT HAS A GOAL OF MIND-CONTROL AND WILL BE USED AGAINST OUR 99% WE THE PEOPLE.
It could have been a RAISING OF ENERGY OF ALL HEAD implants---COCHLEAR, SINUS/NOSE/RETINAL which created what I called BODY ELECTRIC.
I was thinking I was feeling electrical release from cameras and microphones inside my LIVING SPACE not knowing about these IMPLANTS.
03/09/2018
The Color Of Music
Around four percent of the world’s population has some form of synesthesia, a neurological phenomenon that blurs some of the lines around the senses. In two of the more common variants, synesthetes may involuntarily associate letters with colors, or see colors for musical notes—but there are many other forms of synesthesia, all involving the crossover of one form of perception to another.
Writing this week in the Proceedings of the National Academy of Sciences, researchers report that they’ve identified several regions of the genome that may be involved in synesthesia. The team, based at the Max Planck Institute in the Netherlands, examined the genomes of several people in three different families with histories of synesthesia. Within each family, they found gene differences in regions dealing with the formation of axons in the brain during early childhood—though the specific gene variants involved were different from family to family. The work supports the idea that synesthesia may be the result of additional or crossed wiring in certain brain regions.
Amanda Tilot, one of the authors of the study, and Ed Hubbard, an educational psychologist, join Ira to discuss the work, what it implies, and how it fits into what’s already known about this perceptual ability.
Find more information on Tilot’s synesthesia study and to learn how to participate here.
Interview Highlights
On why synesthetes see different shades of colors when listening to musical notes.
Ed Hubbard: Our thinking about this is that some of the same brain processes that are involved in say imagining the music as being dark might be present both in people who experience synesthesia and in all the rest of us that don’t experience synesthesia. But in people who have synesthesia, these processes are heightened or exaggerated in such a way that they do this involuntarily. They often describe it as something that happens to them as opposed to something that they do, and they report that it’s been that way for as long as they can remember, [or] ever since childhood. So it’s quite different at the level of sort of how it feels to you, that subjective experience. But we think at the brain-level, there might be a sort of continuum between what you and I might do voluntarily and what synesthetes report experiencing.
On the potential causes of synesthesia.
Ed Hubbard: One of the main theories about what’s causing synesthesia is a story of crossed wires in the brain that, for example, brain areas that are involved in recognizing letters and words lie directly adjacent to brain areas that are involved in perceiving colors. About 15 years ago, we put forth this idea that there was some sort of what we called “cross-activation.”
So every time somebody who has synesthesia sees a letter or number, in addition to activating those neurons in the brain that are involved in recognizing letters and numbers, they also activate some of those color cells in the brain. And that’s why they then get this automatic, involuntary additional experience of colors.
On whether someone who doesn’t have synesthesia can induce the effects.
Ed Hubbard: Since the late 1800s really, people have asked that same question. And the answer seems to be kind of. With lots and lots of intensive training, people can learn to have synesthesia-like associations. A recent study has even shown that you get these really nice systematic changes in brain activity along with these changes in people’s report. But it doesn’t seem to be quite as automatic. It doesn’t seem to be quite as stable. And the changes may go away fairly quickly after you stop the training. So there does seem to be something still different between synesthetes and non-synesthetes in that respect, but those of us that are curious about it might find a way to at least get some insight into what it’s like.
On why synesthesia is often difficult to detect.
Amanda Tilot: A lot of people don’t realize that their perceptions are anything unusual until maybe they hear about it in a book or in a neuroscience class in college. So that’s actually a tricky thing. It’s very common, but people don’t talk about their perceptions with their friends and realize that there’s something a little different.
On determining if it’s genetically passed down.
Amanda Tilot: Yes, that’s what we think. And we’ve had some indication that that was the case for about 130 years. So that idea that it runs in families is not new. But it’s been taking a long time to figure out exactly what was happening…
Our next task was to figure out if there were any functions of [the genes that contained changes specific to synesthesia] that would tie them all together. So maybe they’re all different, but they act in similar biological pathways. Maybe they have similar roles, maybe even in the brain. And when we look for that, what activities were overrepresented in this big list that we had from the three families [of people with synesthesia from a continued 10-year study], we found that genes related to how neurons connect with each other during development—how they know where to go to send their connections to link the right circuits together—that was a function that was overrepresented in our list of genes that were showing differences in the synesthetes. And that was surprising and exciting for us.
On how synesthesia might affect learning.
Ed Hubbard: Synesthetes will say that sometimes their colors help them to remember things like phone numbers or math facts. They will also say that sometimes they get in the way when, for example, the colors for three and four don’t mix to create the color that seven should be. Or two plus five, which are different colors, are supposed to give them the same color for the seven.
So synesthetes say it helps them and it hurts them in all sorts of interesting ways. And we’re still trying to understand this. We’re looking at adults, college students here that have synesthesia, and also looking at children trying to understand better how these synesthetic associations help and hurt their learning.
First, we want to remember this----these TELEMEDICINE research and experiments are GLOBAL PRIVATE MILITARY CORPORATE research so the goals are less public health and more DEEP, DEEP REALLY DEEP STATE ----MIND-CONTROL AND SURVEILLANCE.
The goals of RETINAL IMPLANTS are to alter what our BRAINS say we see and what we REALLY see. The retina is where that visual information is sent and translated by the BRAIN. If that visual information is ALTERED at the RETINA----your BRAIN thinks it sees something different.
'The retina also contains the nerves that tell the brain what the photoreceptors are "seeing."'
The second concern about RETINAL IMPLANTS has to do with using FULL-SIGHTED PEOPLE like ME -----as CONTROLS -----not only in manipulating color and sight but in using visuals for CODING AND SIGNS tied to spying.
'Many of these are packed into the fovea, a small pit in the back of the eye that helps with the sharpness or detail of images'.
For me as a FULL-SIGHTED human having RETINAL IMPLANT illegally and without consent---installed my concern is this: These implants emit radiation and they are installed very near what is called the FOVIA where all RODS AND CONES exist. Given the numbers of electrodes connected to a RETINAL IMPLANT-----the deterioration of these RODS AND CONES would be HASTENED.
In a FULL-SIGHTED human like me----there would be no loss of color vision during a normal lifetime. Exposure to RETINAL IMPLANT EXPERIMENTATION places FULL-SIGHTED HUMANS LIKE ME----at risk of becoming that DISEASE VECTOR for vision---
PIGMENTOSIS AND MACULAR DEGENERATION.
'Factors Affecting Perceptual Threshold in Argus II Retinal ...www.ncbi.nlm.nih.gov/pmc/articles/PMC3763895When analyzing data from all subjects who have a range of light threshold levels, we found that 90.3% of electrodes placed in direct contact with the retina and within 3 mm of fovea centralis had thresholds below 1 mC/cm 2, and 80.9% had thresholds below 0.35 mC/cm 2. Therefore, while light threshold measurement can be used by clinicians and ...'
The one effect of having this RETINAL IMPLANT for about 8 years is a loss of SHARPNESS AND DETAIL in that LEFT EYE.
Below we see the factors which cause a human with HEALTHY EYES forced to have these implants slowly lose their sight:
1) An increased sensitivity to LIGHT coming through pupil. I could feel my PUPIL DILATION being controled by these IMPLANTS. These RETINAL IMPLANTS work best with MORE LIGHT coming into PUPIL ergo, they manipulated my PUPIL to open wider the normal. When this happens that increased LIGHT on my RODS AND CONES damage those RODS AND CONES over time.
2) The thermo----or heat factor from radiation emitted from ELECTRODES----also damage the RODS AND CONES over time.
Retinal safety of near infrared radiation in photovoltaic restoration of sight
H. Lorach,1,2,* J. Wang,1,2 D. Y. Lee,2,3 R. Dalal,2
P. Huie,1,2 and D. Palanker1,21Hansen Experimental Physics Laboratory, Stanford University, Stanford, CA 94305, USA 2Department of Ophthalmology, Stanford University, Stanford, CA 94305, USA 3Department of Ophthalmology, Gachon University, Gil Medical Center, Incheon, 21565, South Korea *henri.lorach@gmail.com
Abstract: Photovoltaic restoration of sight requires intense near-infrared light to effectively stimulate retinal neurons. We assess the retinal safety of such radiation with and without the retinal implant. Retinal damage threshold was determined in pigmented rabbits exposed to 880nm laser radiation. The 50% probability (ED50) of retinal damage during 100s exposures with 1.2mm diameter beam occurred at 175mW, corresponding to a modeled temperature rise of 12.5°C. With the implant, the same temperature was reached at 78mW, close to the experimental ED50 of 71mW. In typical use conditions, the retinal temperature rise is not expected to exceed 0.43°C, well within the safety limits for chronic use. ©2015 Optical Society of America
1. Introduction
Electrical stimulation of the retina allows restoration of visual perception in patients blinded by retinal degeneration
[1, 2]. Photovoltaic arrays allow a completely wireless and modular design of the implant, thereby greatly simplifying the implantation procedure [3–5]. In this approach, photodiodes convert incoming light into electric current in each pixel. Since ambient light levels are too low for generating currents sufficient for neural stimulation, more intense light has to be used. To avoid photophobia in patients with remaining light sensitivity, we utilize invisible near-infrared (NIR) wavelengths (about 880nm). To produce charge-balanced pulses of electric current, the NIR light is pulsed. Previous studies demonstrated that NIR (880nm-915nm) light elicits retinal responses to photovoltaic stimulation with irradiance ranging from 0.1 to 5mW/mm2, and pulse durations of 1 to 10ms [4, 5].
'One of the factors defining the safety limits of photovoltaic restoration of sight is the retinal heating due to light absorption, primarily in melanin (RPE and choroid) and in the silicon implant itself. The light intensity is limited by both the ocular laser safety standards (ISO 60825 and ISO 15004) and the thermal safety standards for active implantable medical devices (AIMD) (ISO 14708-1:2014 / EN 45502-1:1997). The first one defines the maximum permissible power that can enter the eye for a specific wavelength, beam size and exposure duration, whereas the second one defines a maximum temperature on the surface of an implant. Since our design involves millisecond pulses of NIR light, both safety standards are based on thermal considerations, as opposed to shorter wavelengths involving phototoxicity, or much shorter pulses (<ns), potentially involving photomechanical effects. Laser safety standards have been developed for normal eyes, but the presence of a silicon implant increases the light absorption, which will therefore decrease the maximum permissible exposure'.
Fig. 3. Retinal damage above the implants after irradiation with increasing power.
(A) Implanted retina before irradiation. (B) In the same retina, no damage can be seen at 60mW, but mild whitening is detectable at 90mW (C), pointed by the yellow arrow. (D) Severe damage and detachment occurs at 125mW.
Fig. 4. Temperature rise in the retina.
A) Spatial distribution of the temperature rise in the rabbit eye at the end of the 100s long irradiation. The temperature rise is maximum at RPE, and it rapidly drops with distance. Small absorption of the 880nm radiation in water results in slight elevation of temperature on the beam axis. B) Comparison of the thermal modeling to direct temperature measurements. Optoacoustic measurements of the RPE temperature rise during 810nm illumination by a 2mm wide beam of 129mW (dashed lines, OA1 and OA2) are plotted along the RPE temperature computed with our model using the same beam characteristics (blue curve). Experimental data replotted from (Kandulla, Elsner et al. 2006) with permission. C) Temperature rise as a function of power (dash line) plotted along with the probability of retinal damage (solid line) for the normal (red) and implanted (green) retina. At 175mW, corresponding to the ED50 in normal retina, the temperature rise reaches 12.5°C. With an implant, the same temperature is reached at 78mW (intersection between green and yellow dash lines), matching the experimental thresholds (solid green curve) with ED50 at 71mW.
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I estimate exposure to RETINAL IMPLANTS for about 8 years and do not seem to have lost COLOR ---but my left eye is losing SHARPNESS AND DETAIL. If this RETINAL IMPLANT is left in my LEFT EYE for another 10 years I may well have MACULAR DEGENERATION---maybe earlier.
This will have been INDUCED DELIBERATELY-------and not a NATURAL DECLINE of eyesight. Below we the normal health of MACULAR HEALTH is 90 years old. At this point as a 62 year old woman---if this RETINAL IMPLANT remains in place I will have the eyes of that 90 YEAR OLD WOMAN.
'In maculas of older adults lacking grossly visible drusen and pigmentary change (i.e., they do not have ARM), the number of cones in the cone-dominated part of the macula is stable at approximately 32,000 through the ninth decade'.
The HEALTHY humans forced and largely unaware they have these RETINAL IMPLANTS are well on the way to having HOSTING SERVER NOSY NEIGHBORS KILL THEIR EYESIGHT.
retinal degeneration, retinitis pigmentosa
Transl Vis Sci Technol. 2013 Apr; 2(4): 1.
Published online 2013 Apr 12. doi: 10.1167/tvst.2.4.1
PMCID: PMC3763895
PMID: 24049718
Factors Affecting Perceptual Threshold in Argus II Retinal Prosthesis Subjects
A. K. Ahuja,1,3 J. Yeoh,2 J. D. Dorn,1 A. Caspi,1 V. Wuyyuru,1 M. J. McMahon,1 M. S. Humayun,3 R. J. Greenberg,1 and L. daCruz2, Argus II Study
Abstract
Purpose
The Argus II epiretinal prosthesis has been developed to provide partial restoration of vision to subjects blinded from outer retinal degenerative disease. Participants were surgically implanted with the system in the United States and Europe in a single arm, prospective, multicenter clinical trial. The purpose of this investigation was to determine which factors affect electrical thresholds in order to inform surgical placement of the device.
Methods
Electrode–retina and electrode–fovea distances were determined using SD-OCT and fundus photography, respectively. Perceptual threshold to electrical stimulation of electrodes was measured using custom developed software, in which current amplitude was varied until the threshold was found. Full field stimulus light threshold was measured using the Espion D-FST test. Relationships between electrical threshold and these three explanatory variables (electrode–retina distance, electrode–fovea distance, and monocular light threshold) were quantified using regression.
Results
Regression analysis showed a significant correlation between electrical threshold and electrode–retina distance (R2 = 0.50, P = 0.0002; n = 703 electrodes). 90.3% of electrodes in contact with the macula (n = 207) elicited percepts at charge densities less than 1 mC/cm2/phase. These threshold data also correlated well with ganglion cell density profile (P = 0.03). A weaker, but still significant, inverse correlation was found between light threshold and electrical threshold (R2 < 0.52, P = 0.01). Multivariate modeling indicated that electrode–retina distance and light threshold are highly predictive of electrode threshold (R2 = 0.87; P < 0.0005).
Conclusions
Taken together, these results suggest that while light threshold should be used to inform patient selection, macular contact of the array is paramount.
Translational Relevance
Reported Argus II clinical study results are in good agreement with prior in vitro and in vivo studies, and support the development of higher-density systems that employ smaller diameter electrodes. (clinicaltrials.gov identifier: NCT00407602)
Keywords: retinal prosthesis, retinal degeneration, retinitis pigmentosa
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As a VICTIM of illegal, unconsented RETINAL IMPLANT experiments I KNOW these implants are on the RETINAL NERVE-----there are three positions one being right on the FOVIA-----if I have that implant RIGHT ON MY FOVIA----those RODS AND CONES will be damaged FASTER ------but, each placement does harm. Placement on the OPTIC NERVE degrades not only the RETINA but that VITAL OPTIC NERVE which is the source of AUTONOMOUS CONTROL OF EYE.
If the OPTIC NERVE deteriorate or is damaged------your eye will not automatically DILATE---it will not automatically PROCESS THE IMAGE as it normally is sent to the BRAIN.
IF THE GOAL IS TO CONTROL WHAT WE SEE-------GLOBAL BANKING 1% WOULD WANT TO KILL THAT OPTIC NERVE SO THEY COULD CREATE A VISUAL MESSAGE TO BE SENT TO THE BRAIN.
Those PARTICIPANTS already BLIND ----are seeing these MESSAGES created by global banking 1% changed before they are sent to the BRAIN. Is the ROD AND CONE and damaged eye of blind being repaired---or are they simply being SENT MESSAGES of VIRTUAL REALITY------manufactured visual sight.
Today, I still have NORMAL VISION---on the left-------while HOSTING SERVER NOSY NEIGHBORS are saying I have PIGMENTOSA. That may be true soon,.
What Is Retinitis Pigmentosa?
Articles On
What Is Retinitis? Retinitis pigmentosa (RP) is a term for a group of eye diseases that can lead to loss of sight. What they have in common is a coloring your doctor sees when he looks at your retina -- a bundle of tissue at the back of your eye. When you have RP, cells in the retina called photoreceptors don’t work the way they're supposed to, and over time, you lose your sight.
It’s a rare disorder that’s passed from parent to child. Only 1 out of every 4,000 people get it. About half of all people with RP have a family member who also has it.
The retina has two types of cells that gather light: rods and cones. The rods are around the outer ring of the retina and are active in dim light. Most forms of retinitis pigmentosa affect the rods first. Your night vision and your ability to see to the side -- peripheral vision -- go away.
Cones are mostly in the center of your retina. They help you see color and fine detail. When RP affects them, you slowly lose your central vision and your ability to see color.
Symptoms
Retinitis pigmentosa usually starts in childhood. But exactly when it starts and how quickly it gets worse varies from person to person. Most people with RP lose much of their sight by early adulthood. Then by age 40, they are often legally blind.
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Let's go back to NOSY NEIGHBORS AND THE GANG and the PSYCHO-SEXUAL TORTURE since JAN 2019. The goals of HITTING me at this point as we say was to UPGRADE these NEW BRAND IMPLANT DEVICES and BATTERIES.
The goal was to make me appear CRAZY---MENTALLY ILL ---DANGEROUS----NEEDING TO BE COMMITTED.
The COCHLEAR IMPLANT receiving FEEDBACK MESSAGES included discussion of my being AUTISTIC-----of my being SYNESTHESIA----able to see numbers and words AS COLOR. Now, I have always been CREATIVE AND A SCIENTIST who does look closely and does appreciate COLOR, TEXTURE, AND Chiaroscuro.
Never did I feel an intensity of these color sensations as was felt during PSYCHO-SEXUAL TORTURE. This is when I was saying I felt MY BODY ELECTRIC with my body feeling an AURA.
What I was feeling was a heightened stimulation of RETINAL IMPLANT directed at RODS AND CONES combined with increased RADIATION ELECTRICAL IMPULSES. The messaging coming to COCHLEAR IMPLANT makes one think you really are A AUTISTIC ASPERGER seeing in color.
All that happens inside your eye is increased stimulation of CONES ---for color ---then RODS for chiaroscuro---shades of gray. That increased intensity creates THERMAL ELECTRICAL EMISSION which makes the BRAIN feel it has an ELECTRICAL AURA.
So, while I was simply living my normal life HOSTING SERVER NOSY NEIGHBORS were saying
SHE IS AUTISTIC---THIS IS WHY SHE IS IN THIS STUDY.
This was ONE reason I was supposedly about to be sent to PSYCH WARD.
There was an increased intensity to sensations I already had.
We did not find results for:
Is synesthesia common among those with autism and Asperger syndrome? 15 Answers Zem Jones Zem Jones, works at Higher Education Answered Sep 27 2014 · Author has 845 answers and 780.4k answer views
Originally Answered:
Is synesthesia common among those with autism and Asperger's?
Almost all the people I know with synaesthesia are on the autism spectrum - I know a lot of people with synaesthesia and only a couple of them do not have an ASC diagnosis but they do have other comorbid conditions such as dyslexia.
My daughter has the strongest levels I have come across; she sees sound in colour, she "sees" people as having colours, almost like auras, her pain is described in colour (she has pain amplification - her doctors think it may be because of the way her senses cross over - and uses visualisation of the oposite colour to her pain to control it), she feels the colour of touch and texture..... Synaesthesia can cause problems and confusions, especially when people use synaesthetic language to describe things that non-synaesthetics don't experience in the same way - my daughter was learning music using a colour based system and almost fell out with her teacher, who had designed the system, because the colours weren't right. But it can be a huge boon too - many synaesthetes are highly creative in art and music, one person I know designed an intuitive data base system based on synaesthetic principles that is likely to change the way we access databases. The world for a synaesthete is a rich and vibrant place, and generally the experience of synaesthesia is very positive..
Is synesthesia common among those with autism and Asperger syndrome?
Zem Jones, works at Higher Education
Originally Answered: Is synesthesia common among those with autism and Asperger's?
Almost all the people I know with synaesthesia are on the autism spectrum - I know a lot of people with synaesthesia and only a couple of them do not have an ASC diagnosis but they do have other comorbid conditions such as dyslexia.
My daughter has the strongest levels I have come across; she sees sound in colour, she "sees" people as having colours, almost like auras, her pain is described in colour (she has pain amplification - her doctors think it may be because of the way her senses cross over - and uses visualisation of the oposite colour to her pain to control it), she feels the colour of touch and texture.....
Synaesthesia can cause problems and confusions, especially when people use synaesthetic language to describe things that non-synaesthetics don't experience in the same way - my daughter was learning music using a colour based system and almost fell out with her teacher, who had designed the system, because the colours weren't right.
But it can be a huge boon too - many synaesthetes are highly creative in art and music, one person I know designed an intuitive data base system based on synaesthetic principles that is likely to change the way we access databases. The world for a synaesthete is a rich and vibrant place, and generally the experience of synaesthesia is very positive.
____________________________
Let's go back to NOSY NEIGHBORS AND THE GANG and the PSYCHO-SEXUAL TORTURE since JAN 2019. The goals of HITTING me at this point as we say was to UPGRADE these NEW BRAND IMPLANT DEVICES and BATTERIES.
The goal was to make me appear CRAZY---MENTALLY ILL ---DANGEROUS----NEEDING TO BE COMMITTED.
The COCHLEAR IMPLANT receiving FEEDBACK MESSAGES included discussion of my being AUTISTIC-----of my being SYNESTHESIA----able to see numbers and words AS COLOR. Now, I have always been CREATIVE AND A SCIENTIST who does look closely and does appreciate COLOR, TEXTURE, AND Chiaroscuro.
Never did I feel an intensity of these color sensations as was felt during PSYCHO-SEXUAL TORTURE. This is when I was saying I felt MY BODY ELECTRIC with my body feeling an AURA.
What I was feeling was a heightened stimulation of RETINAL IMPLANT directed at RODS AND CONES combined with increased RADIATION ELECTRICAL IMPULSES. The messaging coming to COCHLEAR IMPLANT makes one think you really are A AUTISTIC ASPERGER seeing in color.
All that happens inside your eye is increased stimulation of CONES ---for color ---then RODS for chiaroscuro---shades of gray. That increased intensity creates THERMAL ELECTRICAL EMISSION which makes the BRAIN feel it has an ELECTRICAL AURA.
So, while I was simply living my normal life HOSTING SERVER NOSY NEIGHBORS were saying
SHE IS AUTISTIC---THIS IS WHY SHE IS IN THIS STUDY.
This was ONE reason I was supposedly about to be sent to PSYCH WARD.
There was an increased intensity to sensations I already had.
'youtube.com
Body Electric (LYRICS) - Lana Del Rey
*All materials used in this video are property of their respective owners.…'
I kept saying I was FEELING THE BODY ELECTRIC while these RETINAL IMPLANTS were hitting my RETINA AND OPTICAL NERVE with higher electrical impulses aimed at CONES----COLOR giving me a feeling of ELATION.
This is much like the 1960s when global banking 1% sold LSD----TAKING A TRIP as a mind-altering FAD.
PLEASE DO NOT THINK GETTING THESE RETINAL IMPLANTS WILL ENHANCE YOUR LIFE OR ABILITY TO GET A JOB----IT HAS A GOAL OF MIND-CONTROL AND WILL BE USED AGAINST OUR 99% WE THE PEOPLE.
It could have been a RAISING OF ENERGY OF ALL HEAD implants---COCHLEAR, SINUS/NOSE/RETINAL which created what I called BODY ELECTRIC.
I was thinking I was feeling electrical release from cameras and microphones inside my LIVING SPACE not knowing about these IMPLANTS.
03/09/2018
The Color Of Music
Around four percent of the world’s population has some form of synesthesia, a neurological phenomenon that blurs some of the lines around the senses. In two of the more common variants, synesthetes may involuntarily associate letters with colors, or see colors for musical notes—but there are many other forms of synesthesia, all involving the crossover of one form of perception to another.
Writing this week in the Proceedings of the National Academy of Sciences, researchers report that they’ve identified several regions of the genome that may be involved in synesthesia. The team, based at the Max Planck Institute in the Netherlands, examined the genomes of several people in three different families with histories of synesthesia. Within each family, they found gene differences in regions dealing with the formation of axons in the brain during early childhood—though the specific gene variants involved were different from family to family. The work supports the idea that synesthesia may be the result of additional or crossed wiring in certain brain regions.
Amanda Tilot, one of the authors of the study, and Ed Hubbard, an educational psychologist, join Ira to discuss the work, what it implies, and how it fits into what’s already known about this perceptual ability.
Find more information on Tilot’s synesthesia study and to learn how to participate here.
Interview Highlights
On why synesthetes see different shades of colors when listening to musical notes.
Ed Hubbard: Our thinking about this is that some of the same brain processes that are involved in say imagining the music as being dark might be present both in people who experience synesthesia and in all the rest of us that don’t experience synesthesia. But in people who have synesthesia, these processes are heightened or exaggerated in such a way that they do this involuntarily. They often describe it as something that happens to them as opposed to something that they do, and they report that it’s been that way for as long as they can remember, [or] ever since childhood. So it’s quite different at the level of sort of how it feels to you, that subjective experience. But we think at the brain-level, there might be a sort of continuum between what you and I might do voluntarily and what synesthetes report experiencing.
On the potential causes of synesthesia.
Ed Hubbard: One of the main theories about what’s causing synesthesia is a story of crossed wires in the brain that, for example, brain areas that are involved in recognizing letters and words lie directly adjacent to brain areas that are involved in perceiving colors. About 15 years ago, we put forth this idea that there was some sort of what we called “cross-activation.”
So every time somebody who has synesthesia sees a letter or number, in addition to activating those neurons in the brain that are involved in recognizing letters and numbers, they also activate some of those color cells in the brain. And that’s why they then get this automatic, involuntary additional experience of colors.
On whether someone who doesn’t have synesthesia can induce the effects.
Ed Hubbard: Since the late 1800s really, people have asked that same question. And the answer seems to be kind of. With lots and lots of intensive training, people can learn to have synesthesia-like associations. A recent study has even shown that you get these really nice systematic changes in brain activity along with these changes in people’s report. But it doesn’t seem to be quite as automatic. It doesn’t seem to be quite as stable. And the changes may go away fairly quickly after you stop the training. So there does seem to be something still different between synesthetes and non-synesthetes in that respect, but those of us that are curious about it might find a way to at least get some insight into what it’s like.
On why synesthesia is often difficult to detect.
Amanda Tilot: A lot of people don’t realize that their perceptions are anything unusual until maybe they hear about it in a book or in a neuroscience class in college. So that’s actually a tricky thing. It’s very common, but people don’t talk about their perceptions with their friends and realize that there’s something a little different.
On determining if it’s genetically passed down.
Amanda Tilot: Yes, that’s what we think. And we’ve had some indication that that was the case for about 130 years. So that idea that it runs in families is not new. But it’s been taking a long time to figure out exactly what was happening…
Our next task was to figure out if there were any functions of [the genes that contained changes specific to synesthesia] that would tie them all together. So maybe they’re all different, but they act in similar biological pathways. Maybe they have similar roles, maybe even in the brain. And when we look for that, what activities were overrepresented in this big list that we had from the three families [of people with synesthesia from a continued 10-year study], we found that genes related to how neurons connect with each other during development—how they know where to go to send their connections to link the right circuits together—that was a function that was overrepresented in our list of genes that were showing differences in the synesthetes. And that was surprising and exciting for us.
On how synesthesia might affect learning.
Ed Hubbard: Synesthetes will say that sometimes their colors help them to remember things like phone numbers or math facts. They will also say that sometimes they get in the way when, for example, the colors for three and four don’t mix to create the color that seven should be. Or two plus five, which are different colors, are supposed to give them the same color for the seven.
So synesthetes say it helps them and it hurts them in all sorts of interesting ways. And we’re still trying to understand this. We’re looking at adults, college students here that have synesthesia, and also looking at children trying to understand better how these synesthetic associations help and hurt their learning.
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