'There is no specific cause of ankylosing spondylitis, but there are autoimmune and/or genetic triggers identified in some people'
The subliminal messaging happening during this period----2010----2015 I think was the cause of my constantly repeating verbage like I AM HOT I AM COLD I AM SLEEPY/TIRED.
I never said those things before and I realized right after saying these things ---that I was NOT HOT NOT COLD NOT SLEEPY/TIRED.
This doesn't mean there was NO BODY TEMPERATURE CONTROL. It was significant if there was.
Another variable in this was MY JOINTS feeling stiff. I have indeed felt a lot joint stiffness these several years and I have explained it by saying---ARTHRITIS RUNS IN MY FAMILY.
I always think back to when my mother started to feel these symptoms. She was in mid-60s and I am heading for mid-60s but those joint pains did start early.
JOINT PAIN AND STIFFNESS IN FINGER JOINTS----IN KNEES AND THE ANKLE PAIN CAME FROM MY LEG INJURY.
Are Thyroid Disease and Arthritis Connected?
Was I feeling NECK STIFFNESS-----is my bad posture from IMPLANT manipulation-----I think not---I have always had pain in my neck because I have a long cervical spine and have always not held my shoulders back----sorry, NO BEAUTY SCHOOL FOR ME.
As I said earlier-----MY HEART BEAT was never manipulated ----the microphones attached to COCHLEAR IMPLANTS were making LOUD SOUNDS of my heart beat.
Thyroid Gland: Overview
How this endocrine-realted thyroid gland functions, and what symptoms might be a clue for hyperthyroidism and hypothyroidism
Written by Bridget Brady MD, FACS
The thyroid gland is a butterfly-shaped organ located in the base of your neck. It releases hormones that control metabolism—the way your body uses energy. The thyroid's hormones regulate vital body functions, including:
- Heart rate
- Central and peripheral nervous systems
- Body weight
- Muscle strength
- Menstrual cycles›
- Body temperature
- Cholesterol levels
- Much more!
The thyroid gland is about 2-inches long and lies in front of your throat below the prominence of thyroid cartilage sometimes called the Adam's apple. The thyroid has two sides called lobes that lie on either side of your windpipe, and is usually connected by a strip of thyroid tissue known as an isthmus. Some people do not have an isthmus, and instead have two separate thyroid lobes.
How the Thyroid Gland Works
The thyroid is part of the endocrine system, which is made up of glands that produce, store, and release hormones into the bloodstream so the hormones can reach the body's cells. The thyroid gland uses iodine from the foods you eat to make two main hormones:
- Triiodothyronine (T3)
- Thyroxine (T4)
The hypothalamus produces TSH Releasing Hormone (TRH) that signals the pituitary to tell the thyroid gland to produce more or less of T3 and T4 by either increasing or decreasing the release of a hormone called thyroid stimulating hormone (TSH).
- When T3 and T4 levels are low in the blood, the pituitary gland releases more TSH to tell the thyroid gland to produce more thyroid hormones.
- If T3 and T4 levels are high, the pituitary gland releases less TSH to the thyroid gland to slow production of these hormones.
Why You Need a Thyroid Gland
T3 and T4 travel in your bloodstream to reach almost every cell in the body. The hormones regulate the speed with which the cells/metabolism work. For example, T3 and T4 regulate your heart rate and how fast your intestines process food. So if T3 and T4 levels are low, your heart rate may be slower than normal, and you may have constipation/weight gain. If T3 and T4 levels are high, you may have a rapid heart rate and diarrhea/weight loss.
Listed below are other symptoms of too much T3 and T4 in your body (hyperthyroidism):
- Irritability or moodiness
- Nervousness, hyperactivity
- Sweating or sensitivity to high temperatures
- Hand trembling (shaking)
- Hair loss
- Missed or light menstrual periods
- Trouble sleeping
- Tiredness and fatigue
- Difficulty concentrating
- Dry skin and hair
- Sensitivity to cold temperature
- Frequent, heavy periods
- Joint and muscle pain
HOSTING SERVER NOSY NEIGHBORS are working on SPACE HUMAN HIBERNATION AND TRAVEL. This would involve control SLEEP----controlling HEART BEAT----controlling BODY TEMPERATURE. HOSTING SERVER NOSY NEIGHBORS tried hard to PRETEND they were manipulating these body functions in me ----but I DO NOT BELIEVE THAT. They disrupted my SLEEP PATTERNS which were normal with constant attack of MY NERVES-----TOOTH PAIN from NOSE IMPLANT hitting the mouth-----SHOULDER PAIN by hitting the body center nerves near the cranial nervous system----LEG CRAMPING severe not tied to my leg injury by HITTING that LEG IMPLANT.
HOSTING SERVER NOSY NEIGHBORS constantly say-----SHE IS GOING TO BED EARLIER AND EARLIER----WE ARE PUTTING HER INTO A DEEP SLEEP----HIBERNATION.
That is FAKE NEWS FAKE DATA-----they were simply making my body PAIN keep me awake.
The other goal of HUMAN HIBERNATION is getting humans to EAT LESS-----I was EATING MORE. I was eating more because I am a POST-MENOPAUSAL WOMAN who has a natural craving for sweets----and not caring about my SLIM MUSCULAR FIGURE.
Subliminal messaging by global banking 1% these few decades is the TOP cause of WEIGHT GAIN or WEIGHT disease vectors like bulimia there is. No GLANDULAR manipulation needed.
The goals of HOSTING SERVER NOSY NEIGHBORS was to CONTROL me----that would mean they needed to have my WEIGHT under control and they didn't. I gained weight even while I remember saying ----I AM LITHE----I AM LITHE. I never used that word before so I was being given subliminal messaging which DIDN'T WORK.
HOSTING SERVER NOSY NEIGHBORS SAID IN FEEDBACK ----SHE IS WANTING TO BE LITHE FOR ALL THE MEN WATCHING HER.
'Lithe Synonyms, Lithe Antonyms | Thesaurus.comwww.thesaurus.com/browse/lithe
Lithe, full-bosomed, and ruddy, she radiated a powerful and subtle charm. He was broad of shoulder with erect, military figure; while they were lithe and sinewy. Then he noticed that she was tall and lithe, and that she had fine brown eyes and hair. He was lithe, of indeterminate age, but with a look of great determination'.
HOSTING SERVER NOSY NEIGHBORS loved to call people captured by ILLEGAL, CRIMINAL IMPLANTS AND 24/7 video PORN -----
The other body function HOSTING SERVER NOSY NEIGHBORS want to control for HUMAN HIBERNATION is BREATHING----and BODY TEMPERATURE. This is why there was constant FEEDBACK saying they were making it hard for me to breath----when of course that was the NOSE IMPLANT filling my nose and lungs WITH MUCUS.
The sensitivity to temperature has to do with AGE---as we age our BLOOD GETS THINNER as they say. I have always been a little chilled maybe because I have LOW HEMOGLOBIN-----RED BLOOD CELLS----ANEMIA.
People with anemia tend to feel CHILLED.
For ME------I was not experiencing through BRAIN manipulation ANY of these body function controls. Lots of NERVE/JOINT PAIN-----
Human hibernation: Secrets behind the big sleep
The extreme survival tricks of hibernating animals… and the occasional human… could help us overcome life-threatening injuries, as Frank Swain discovers.
By Frank Swain 5th May 2014 I
Imagine it: you have been rushed into the emergency room and you are dying. Your injuries are too severe for the surgeons to repair in time. Your blood haemorrhages unseen from ruptured vessels. The loss of blood is starving your organs of vital nutrients and oxygen. You are entering cardiac arrest.
But this is not the end. A decision is made: tubes are connected, machines whir into life, pumps shuffle back and forth. Ice-cold fluid flows through your veins, chilling them. Eventually, your heart stops beating, your lungs no longer draw breath. Your frigid body remains there, balanced on the knife-edge of life and death, neither fully one nor the other, as if frozen in time.
The surgeons continue their work, clamping, suturing, repairing. Then the pumps stir into life, coursing warm blood back into your body. You will be resuscitated. And, if all goes well, you will live.
Suspended animation, the ability to set a person’s biological processes on hold, has long been a staple of science fiction. Interest in the field blossomed in the 1950s as a direct consequence of the space race. Nasa poured money into biological research to see if humans might be placed in a state of artificial preservation. In this state, it was hoped, astronauts could be protected from the dangerous cosmic rays zapping through space. Sleeping your way to the stars also meant carrying far less food, water and oxygen, making the ultimate long-haul flight more practical.
There has long been interest in whether suspended animation could allow astronauts to survive missions to Mars and beyond (Science Photo Library).
One recipient of that funding was a young James Lovelock. The scientist would dunk hamsters into ice baths until their bodies froze. Once he could no longer detect a heartbeat, he would reanimate them by placing a hot teaspoon against their chest (in later experiments, Lovelock warmed to the space-age theme by building a microwave gun out of spare radio parts to more gently revive his test subjects). These experiments on the flexibility of life would set him on the path to his most famous work, the “Gaia hypothesis” of the world as a living super-organism.
Adventurous as they were, these early experiments did not progress beyond the animal stage, and astronauts were never frozen and revived with hot spoons. The idea of transforming people into inanimate bars of flesh for long-distance space travel remained in the realm of science fiction. Nasa’s interest tailed off with the end of the space race, but the seeds planted by Lovelock and his colleagues continued to grow.
In 1900, the British Medical Journal published an account of Russian peasants who, the author claimed, were able to hibernate. Existing in a state approaching “chronic famine”, residents of the north-eastern Pskov region would retreat indoors at the first sign of snow, and there gather around the stove and fall into a deep slumber they called “lotska”. Waking once a day to wash some hard bread down with water, the family took it in turns to watch the fire, only rousing themselves fully once spring had broken. No trace of the sleepy peasants of Pskov has ever emerged since, but the fantasy of human hibernation persists, and very occasionally, something that looks very similar to it crosses into reality.
A century later, Anna Bagenholm was on a skiing holiday in Norway when she crashed head first into a frozen stream and became trapped under the ice. When rescuers finally arrived, the Swedish radiologist had been submerged for 80 minutes, and her heart and breathing had stopped. Doctors at Tromso University Hospital recorded a body temperature of 13.7C, the lowest ever observed in a victim of accidental hypothermia. By all accounts she appeared to have drowned. And yet, after careful rewarming and ten days spent in intensive care, Bagenholm woke up. She went on to recover almost fully from her cold brush with death. Under normal circumstances, even a few minutes trapped underwater would be enough to drown a person, and yet Bagenholm had survived for over an hour. Somehow the cold had preserved her.
It’s not the first time the benefits of cold for traumatic injury have been made apparent. As far back as the Napoleonic era, medics noted that wounded infantrymen left out in the cold had better survival rates than the wounded officers kept close to the fire in warmed tents. Therapeutic hypothermia is now commonly used in hospitals to reduce injury in a wide variety of situations, from surgery to helping infants recuperate following difficult births.
Lowering your body temperature slows your metabolic activity, about 5–7% for every degree dropped. This in turn reduces the rate at which you consume essential nutrients such as oxygen. Tissues that might become starved of oxygen due to blood loss or cardiac arrest are thus protected. In theory, if we were to keep reducing your temperature, eventually your biological processes would come to a standstill. You would exist in a state of suspended animation. Like a stopped clock, there’d be nothing physically wrong with you – all the components inside would still be intact, simply stationary. All it would take would be a little heat to set you in motion again.
THERE IS NO BASIC SCIENCE SAYING THIS HIBERNATION CAN BE DONE----IT IS ALL BLIND AMBITION.
Of course, it’s not that simple. Hypothermia is dangerous. Your body wants to be warm and will fight to remain that way. Throughout your life, it will maintain a fairly constant temperature of around 37C. This requires great effort. Your body must perform countless constant adjustments to balance heat production with heat lost to the environment, working to keep your temperature within a narrow band. If it drops too low, your blood is shunted away from the exposed skin and gathers in your central torso while you shiver and huddle under blankets. The effects of more severe cold are disastrous. At a body temperature of around 33C – just four degrees below normal – your heartbeat begins to flutter. At 25C, there’s a risk it will stop altogether. And even if you survive hypothermia, warming you up again can cause extensive kidney damage.
Arctic ground squirrels make sure their bodily fluids don’t freeze solid during hibernation (Science Photo Library)
There are, however, certain species of animal that can endure far greater spells of cold. The Arctic ground squirrel normally maintains a body temperature similar to our own. But during hibernation, it can survive a core temperature as low as –3C, carefully managing its super-cooled bodily fluids so that it won’t freeze solid. And Lovelock’s hamsters could survive hypothermic depths that would kill us. How animals survive these states is of great interest to anyone hoping to unlock the secrets of suspended animation for humans.
“When is your comrade dead?” asks Professor Rob Henning with a grin, quoting an Army handbook he received as one of the Netherlands’ last draft of conscripts. “One: Is he rotting? Two: Is his head more than twenty centimetres from his body?” Like Lovelock, Henning has conducted experiments with hibernators that have given him a flexible view of what constitutes being alive.
From the top floor of the Department of Clinical Pharmacy and Pharmacology at the University Medical Centre Groningen (UMCG), a large window looks down on the medieval city spread over a pancake-flat landscape. Below is a bustling hospital, the region’s hub for transplant surgery. It’s also where Henning and his team are uncovering the secrets of hibernation.
“What we’re doing here is biomimicry,” says Henning, “using these great adaptations in nature to hijack them for the benefit of medicine.”
Many animals can slow their metabolism to enter low-energy states: insects, amphibians, mammals, birds and fish. In short periods, this condition – characterised by reduced body temperature and inactivity – is known as torpor. By stringing together many of these short sessions of torpor, animals can enter the long-term dormancy we call hibernation. With this technique, small animals such as mice, hamsters and bats can last out the cold famines of winter huddled away, conserving energy.
Trained as an anaesthetist, Henning started “hobbying” in hibernation in the 1990s, but things took off in earnest when his research group was formed around six years ago. “If you think about hibernators, you have a lot of applications. The most obvious ones are any type of major surgery,” he explains. Blood loss is the major cause of death during surgery, but in their hypothermic state, hibernators can survive far worse injuries than they can at normal body temperatures. This is partly because tissues are protected at low metabolic rates, and partly because the heart is pumping blood at a fraction of the rate it usually does.
A dormouse in torpor. It will spend up to three quarters of its life asleep, hibernating (Science Photo Library)
But a resistance to cold and blood loss isn’t the total sum of hibernators’ incredible endurance. Although it resembles a very long lie-in, hibernating is not a simple matter of sleeping through the cold. It’s a gruelling marathon of hypothermia, starvation and disease susceptibility. To endure these sufferings, the animals that practice it have developed a suite of adaptations to protect mind and body.
Before a long hibernation, animals eat their way into obesity, essentially becoming type 2 diabetic. Unlike in humans, this does not result in the thickening of artery walls that leads to heart disease. Some species will stop eating two or three weeks before hibernation, suddenly resistant to the pangs of hunger even while maintaining their regular level of activity.
While a human can lie in bed for a week before muscles begin to atrophy and blood clots form, hibernators will endure months without moving. During hibernation, the microbiome – the community of bacteria living in an animal’s digestive tract – is battered by cold and the sudden lack of food. Hibernators’ lungs become covered with a thick deposit of mucus and collagen like those seen in people with asthma, and their brains show changes that resemble those of early-stage Alzheimer’s. Some species lose memory during hibernation. Most surprising of all, some show symptoms of sleep deprivation when they finally wake. And yet, hibernators are able to counter all of these issues to bounce back in spring, often without any long-term ill effects.
Thicker than water
UMCG is a half-kilometre complex of buildings so tightly huddled together that it’s possible to walk from the grand foyer at one end to the bicycle racks at the other without stepping outside. One of these buildings is the animal laboratory.
In a tiny room set away from the main corridor, Henning’s doctoral student Edwin de Vrij and his colleague are tending to a rat laid prone on a bed of ice. A tangle of fine tubes and wires surrounds the animal, delivering life-preserving fluids and carrying away precious data. A spool of paper inching from one machine shows that from a frenetic 300 beats per minute, the rat’s heart rate has slowed to just 60. The red numbers glowing on another show that the rat’s internal temperature has dropped more than 20 degrees to 15C. Clicking like a metronome, a ventilator delivers steady breaths to the anaesthetised rodent. As a non-hibernator like us, the rat cannot survive deep hypothermia without medical assistance. “If you cool them down, nerve impulses will be slower, and muscles have a harder time in the cold, so it’s quite physiological that they have a harder time breathing,” explains de Vrij. This isn’t the case for true hibernators – or some other non-hibernating mammals, for that matter. “Somehow hamsters can maintain adequate breathing,” he says. “We don’t have to ventilate them.”
As well as inducing hibernation in hamsters (a process that takes weeks of gradual adjustment in climate-controlled rooms to mimic the onset of winter), the UMCG team also induce forced hypothermia states like that of our rat, chilling the animals rapidly until they fall into a state of metabolic suspension.
Today, de Vrij is searching for platelets, which are essential for blood clotting to prevent bleeding. Hibernating animals avoid getting blood clots despite their lack of activity, an ability that comes down partly to a curious change in the hypothermic body: as they cool, platelets disappear from the blood. Nobody yet knows where they go, but their prompt reappearance on rewarming has de Vrij convinced that they are preserved somewhere in the body, rather than being absorbed and later resynthesised. Surprisingly, this change also happens even in non-hibernators, including rats and – occasionally – human victims of hypothermia.
Platetets (pink) are vital for blood clotting, and could play a key role in helping animals survive during hibernation (Science Photo Library)
The shared characteristics of different hibernators mean it’s likely that these species have inherited fragments of protective mechanisms against cold, inactivity, starvation and asphyxiation from common ancestors and developed these into a comprehensive low-metabolic syndrome. There are even hints that we humans might, to some extent, retain some of these abilities. For a long time, there was no evidence that primates could hibernate. But in 2004, a species of Madagascan lemur was shown to practice regular bouts of torpor. “If you look at the lemur and look at us, we share about 98% of our genes,” says Henning. “It would be very strange if the tools of hibernation were all packed into that 2% difference.”
As their body temperature drops, hibernators also remove the lymphocytes (white blood cells) from their blood and store them in the lymph nodes. And within 90 minutes of awakening, these reappear. This damping down of the immune system prevents a general inflammation in the body during rewarming – the very thing that would cause humans and other non-hibernators to suffer kidney damage. However, it’s a risky strategy, leaving animals unable to mount an immune defence while hibernating. The fungus responsible for white-nose syndrome, currently wiping out bat colonies in the USA, takes advantage of this vulnerability, infecting the bats while they are dormant. In response, the bats frequently exit hibernation and rewarm to fight off the pathogen – the high-energy cost of these interruptions ultimately killing them.
Knowing how hibernators control these changes in their blood could have immediate and far-reaching benefits for us. As well as improving our ability to survive hypothermia and cold suspended-animation states, stripping the blood of white blood cells could prevent the aseptic sepsis caused by heart–lung machines, in which activation of blood cells as they pass through the life-support equipment triggers a body-wide immunological reaction. Transplant organs, often chilled for transport, would also benefit from better cryoprotection. And we could increase the shelf-life of our blood stocks – we still haven’t figured out how to store donated blood platelets at low temperatures, so blood donations can only be kept a week before they must be used or thrown away due to the risk of bacterial infection.
The UMCG team took a giant leap towards achieving these goals quite by accident after a student left a culture of hamster cells in a fridge at 5C. After a week the hamster cells were still alive, and smelling of rotten eggs. The student poured the medium surrounding the cells over a separate batch of cells from a rat, suspecting the smelly cells might have secreted some kind of protective agent. She placed them in the same fridge and waited. Normally, refrigerating rat cells would quickly kill them, but after two days they were still alive.
The team is investigating several compounds that might be responsible for this cryopreservation. One is an enzyme known as cystathionine beta synthase (CBS), which stimulates the production of hydrogen sulphide, the molecule that gives rotten eggs their characteristic whiff. If hamsters are injected with a chemical to inhibit CBS, they can no longer enter torpor, and those that were forced into hypothermic states suffered the kind of kidney damage one would expect in non-hibernators like us.
Of over a hundred compounds Henning’s team has investigated, many had no effect, but a few did, conferring long-term cold protection to cell samples. The team has already patented one of these compounds, Rokepie, as an additive. This would allow cells that normally need to be kept at 37C, such as those from humans or mice, to be stored in the refrigerator, either for transport or so experiments can be put on hold during weekends and busy periods.
The leading cryopreservation molecules extracted from hibernators are incredibly potent, and it seems they work by eliciting changes in the cells themselves – whether these are from hibernators or not. If so, this offers further evidence that we still possess some tools that could help endure hypothermia and low metabolic states.
For now, applying the lessons they’ve learned from hibernators wholesale onto humans is not within the remit of Henning’s group. The space race is long over, and Nasa is not awarding major grants to develop suspended animation. However, the US Army is.
“If you look anywhere near a trauma bay, things are pretty chaotic,” says Professor Sam Tisherman. “It’s controlled chaos, but the chaos mainly comes from the fact you never know what’s going on with the patient.”
In frenetic hospital emergency wards, it’s often not possible for doctors to identify the problem, fix it and keep the patient alive all at the same time. Patients suffering uncontrolled blood loss, for example, may go into cardiac arrest. When this happens, surgeons must fight the clock to stop the bleeding before they can start resuscitation efforts. “Somebody rolls in and they’re basically dying,” says Tisherman. “We’re quickly trying to resuscitate them, and figure out what’s wrong with them, and repair injuries all at the same time.” This is the fundamental underpinning of trauma medicine: you are always against the clock.
Tisherman wants to buy doctors a little more time. He believes that by inducing hypothermia we can extend the “golden hour” in which surgeons battle to save the lives of critically injured patients. To do this, he’s pushing human endurance of hypothermia far beyond its normal limits.
After graduating from MIT in 1981, Tisherman built a career in critical care medicine. He won a Lifetime Achievement Award in Trauma Resuscitation Science from the American Heart Association in 2009, and is now an Associate Director of the Safar Center for Resuscitation Research in Pittsburgh. It was founded by Peter Safar, the Austrian physician who popularised the “kiss of life”, CPR, and drove the creation of the Resusci Anne doll used in teaching it. At Pittsburgh, Safar created the world’s first intensive care training programme. His lifelong aim was to “save the hearts and brains of those too young to die.”
The procedure that Tisherman is pioneering is called emergency preservation and resuscitation. His work is supported through the US Army’s Telemedicine and Advanced Technology Research Center, which funds research on topics as niche as advanced prosthetics and robots to carry wounded soldiers out of the battlefield.
Some of his surgeons will already be familiar with hypothermic techniques, having routinely chilled patients to the low 30s or high 20s. For procedures that require zero blood flow, cardiac surgeons will even cool patients to around 15C, the point at which their heart stops.
Surgeons performing a cardiac operation using hypothermic protection of the patient in Russia in 1979 (Science Photo Library)
Tisherman is planning to cool patients to this point, and perhaps even further, chilling them to such a degree that the entire body enters a kind of suspended animation. During this time, they will have no heartbeat, no breathing and no discernible brain activity. In fact, they’ll have no blood, either – it will be drained and replaced with ice-cold saline, the only way to cool a human fast enough to avoid tissues becoming damaged as they struggle to remain functioning. Tisherman calls this state “hypothermic preservation”.
The procedure has already been demonstrated successfully in the lab, reviving dogs that had lain suspended in cold states for up to three hours. Trials are now moving to a clinical setting. Surgeons, anaesthetists and perfusionists at Massachusetts General Hospital have even undergone training for the pioneering surgery. But no one knows when a suitable patient will arrive through the doors. That is in fact one of the issues they face: by the nature of trauma, patients won’t be able to give informed consent for the procedure. Because of this, Tisherman’s group has engaged in a wide community consultation to let citizens in the area know the programme was going on. The study had to be signed off personally by the Secretary of the Army, the highest-ranking civilian official in the organisation.
Beyond that lie further obstacles. Amid the frantic activity of the emergency room, Tisherman must make sure that a team of trauma surgeons can work in concert with cardiac surgeons and perfusionists armed with pumps and bags of chilled saline, an additional layer of complexity in an already chaotic environment. And although cooling affects all tissues equally, it is not without secondary effects. The blood factors responsible for clotting are also inhibited by the cold. This creates problems controlling bleeding during the rewarming phase. The surgeons, too, will suffer from the cold, as both the patient and the room itself will be chilled during the procedure. Yet the cold is only a tool; the end goal is metabolic suspension.
In the future, emergency preservation and resuscitation could be extended to those suffering heart attacks or exposure to poisons, or any critical care situation where time is a factor. “Cooling is the most powerful way of suppressing metabolism we have,” says Tisherman, “If we can either decrease the needs of the tissues or improve oxygen delivery to the tissues then everything will be okay.”
Although animals in the laboratory were able to recover from three hours in this suspended state, the first human patients to experience it will only be put under for a third of that. “An hour should be enough to repair the bleeding,” Tisherman says. “The cooling period doesn’t necessarily have to cover the entire surgery.” For those wanting to travel to distant stars, going beyond that hour is, sadly, out of the question for now. “We’re not trying to freeze the dead,” Tisherman chuckles, “just buy enough time to save the living.”
HOSTING SERVER NOSY NEIGHBORS say SHE'S GOING DOWN ----meaning they are PRETENDING to control the body functions tied to sending me into an hibernation.
So, I am gaining weight-----they have diagnosed me as BECOMING TYPE 2 DIABETIC-----they are saying my BLOOD CELL counts including PLATELETS are indicating I am going into this HUMAN HIBERNATION.
'Before a long hibernation, animals eat their way into obesity, essentially becoming type 2 diabetic'.
My sugar levels are NORMAL-----they may be a little or low depending upon when I do the blood lab---but, there is no indication of DIABETES.
All my life I have been ANEMIC ---LOW RED BLOOD CELLS. This I address naturally with the foods I eat---
HOSTING SERVER NOSY NEIGHBORS SAY------SHE HAS BLEEDING FROM TOOTH GUMS BECAUSE OF BLOOD CELL COMPROMISE.
I say----HOSTING SERVER NOSY NEIGHBORS are messing with MY THYROID GLAND. Naturally, I have no problem------IMPLANT MANIPULATIONS are creating DISEASE VECTOR symptoms.
'Platetets (pink) are vital for blood clotting, and could play a key role in helping animals survive during hibernation (Science Photo Library)'
Anyone having an BRAIN IMPLANT like RETINAL/NOSE/SINUS will be at the beckon call of those able to control that THYROID---PARATHYROID. The symptoms one may experience are VERY LIKELY NOT NATURAL.
I am HEALING from cuts and injuries JUST FINE.
'Effect of Thyroid Dysfunctions on Blood Cell Count and Red ...www.ncbi.nlm.nih.gov/pmc/articles/PMC3915449
Thyroid dysfunction induces different effects on blood cells such as anemia, erythrocytosis leukopenia, thrombocytopenia, and in rare cases causes’ pancytopenia. It also alter RBC indices include MCV, MCH, MCHC and RDW. Thus this study attempted to evaluate effect of hypo & hyperthyroidism on blood cell count and RBC indices'.
What I hear today on THE NETWORK from NOSY NEIGHBORS is that I am going to die soon from any number of CANCERS. Having these DEVICES inside my body MAY indeed have induced CANCER----but, as likely HOSTING SERVER NOSY NEIGHBORS are MANIPULATING those BLOOD CELL COUNTS to make appear so.
A Link Between a Blood Cancer and Thyroid Disorders
Posted by Thyroid Basics
What is polycythemia vera, and who is at risk of acquiring this rare medical condition?
Polycythemia vera is an iatric disorder that multiplies the number of erythorcytes in a human body. This condition is life-threatening because having an excessive number of red blood cells could result in the viscosity of the blood, reducing blood circulation. This results in blood clots, which may lead to heart failure or stroke. If not treated properly, this disorder could progress to more serious blood cancers, such as acute leukemia.
The disease is known to develop slowly, and the symptoms may not occur evidently. Some people are clueless unless they’re diagnosed by a doctor.
To have ample knowledge of PV, let’s discuss the symptoms which are ordinary yet could be a sign of blood disorder:
- Gum bleeding
- Excessive sweating
- Shortness of breath
- Blurred vision
- Sudden weight loss
- Swelling of joints
How Could a Person Acquire Polycythemia Vera?
In a normal state, your body regulates the number of the blood cells you have – white blood cells, red blood cells, and platelets. However, in polycythemia vera, your bone marrow abnormally produces too many blood cells.
According to a study, PV is developed by the protein switch that commands cells to multiply. Most people who have PV have this kind of protein mutation. This mutation remains uncertain until now, but it has been assured that is not inherited.
Treatment for PV mainly focuses on reducing the number of blood cells. These normally include:
- Low-dose aspirin
- Hydroxyurea (Hydrea)
- Oxygen supplementation
There are studies linking polycythemia vera to thyroid disorders. According to FDA reports, people aged 60 and above who were diagnosed with PV later on acquired hypothyroidism. Most of those diagnosed were men.
A common form of thyroid disorder is hypothyroidism. Hypothyroidism is a disorder where the thyroid gland does not produce enough thyroid hormones. This leads to lower metabolism.
Furthermore, thyroid hormones have a critical role in the formation and proliferation of cells. If thyroid hormones malfunctioned, it could result in production disorder of blood cells.
Those who were diagnosed with PV and acquired hypothyroidism after having some abnormal health conditions. Among these conditions are hyperuricemia (the level of uric acid is abnormally high), high blood pressure, and chronic myeloid leukemia.
According to studies, people who have these diagnoses are taking some drugs to treat the persistent complications. Some of the drugs are:
On the other hand, few patients with hypothyroidism were reported to adopt PV, after that. According to FDA Studies, in this case, women are more likely at risk unlike those who had PV first after acquiring hypothyroidism. They suffer from health conditions such as iron overload, high blood pressure, hypertension, and chronic obstructive pulmonary disease.
Some drugs are also prescribed by doctors to alleviate the treatment. Some of those are:
People diagnosed with the abovementioned blood disorder relating to polycythemia vera are already at high risk to acquire a complex form of their current burden.
This study also shows that people who were suffering from post-hypothyroidism (men) are more likely to survive. Aside from the strong resistance to the side effects of the treatment, the medicines that are made available for them are proven effective. However, people with hypothyroidism and later on polycythemia vera considers the drugs they take ineffective.
When HOSTING SERVER NOSY NEIGHBORS say-----SHE'S DYING-------YES, SHE DOES LIKELY HAVE CANCER-----this is why. The manipulation of the THYROID over several years by BRAIN IMPLANTS can take an AUTONOMOUS system and turn it on its head. Maybe too many RED BLOOD CELLS produced by the THYROID---and someone has a HEART ATTACK. Maybe too many WHITE BLOOD CELLS and someone looks DIABETIC. Maybe too many PLATELETS and someone looks like they have INTERNAL BLEEDING.
My blood labs have indicated I have no blood deficits as of 2010----with a body functioning VERY HEALTHILY.
Between 3 and 5% of all malignant tumors are located in the head and neck region. Approximately half of these are located in the oral cavity, and oral squamous cell carcinoma (OSCC) accounts for roughly 90% of the total'.
BLOOD LEUKEMIA is what FEEDBACK suggests I may have after several years of exposure to IMPLANT radiation and glandular MANIPULATION. This could be true.
Please think about WHEN THE IMPLANT WAS INSTALLED. The battery and microchip technology became more INVASIVE AND DAMAGING with each new product. My IMPLANTS likely 2006--2007---2010----have me exposed for 7 -----9 years MAYBE.
'Radiation can cause cancer in most parts of the body, in all animals, and at any age, although radiation-induced solid tumors usually take 10–15 years, and can take up to 40 years, to become clinically manifest, and radiation-induced leukemias typically require 2–9 years to appear'.
I am pretty sure the BATTERIES and microchips in MY IMPLANTS are going , going , going ----GONE so future damage will STOP. What happens to AUTONOMOUS systems manipulated when these IMPLANT DEVICES are allowed to DIE?
THE AUTONOMOUS IS AUTONOMOUS BECAUSE IT IS HARD-WIRED ---UNLESS THE MID-BRAIN LOBES ARE DAMAGED---AND THEY COULD BE-----THE DEVICE DYING WOULD REVERT TO NORMAL FUNCTIONING.
Any FAKE BODY FUNCTION through manipulation would CORRECT ITSELF-----in a few years.
Dangers of High & Low White Blood Cells + Underlying Factors
Written by Puya Yazdi, MD | Reviewed by Selfhacked Science Team | Last updated: November 11, 2019
Medically reviewed by
Jonathan Ritter, PharmD, PhD (Pharmacology)
White blood cells are immune cells that help fight infections and diseases. Read on to learn what high or low white blood cell counts could mean.
What Are White Blood Cells?
White blood cells, or leukocytes, help the body fight infection. The five types of white blood cells are basophils, neutrophils, eosinophils, lymphocytes, and monocytes .
There are associations between white blood cell (WBC) count and the proteins in red blood cells, heart rate, weight, cholesterol, uric acid, creatinine, sex, ethnic origin, blood pressure, height, and blood sugar in both men and women .
Having a high level of white blood cells is bad, but for some people having too low of a level means that they won’t be able to effectively fight infections.
Why Are Low Levels of White Blood Cells Bad?
Your doctor will interpret your WBC test results, taking into account your medical history and other tests results. A result that is slightly high may not be of medical significance, as this test often varies from day to day and from person to person.
Having too low of a white blood cell count can be a sign of an immune system that is not capable of handling infections properly.
Also, low white blood cells tend to indicate other issues in the body. For example, in some autoimmune conditions, white blood cells can be lower. If you are worried about a low WBC count, work with your doctor to determine and address the underlying causes and develop an appropriate treatment plan.
A low white blood cell count can be caused by the following (not an exhaustive list):
- Infections, including viral infections (especially HIV/AIDS)
- Chronic inflammation, which can use up white blood cells faster than they are produced
- Certain disorders present at birth that involve diminished bone marrow function
- Cancer or other diseases that damage bone marrow, such as leukemia; also, chemotherapy or radiation therapy
- Drugs, such as antibiotics, which destroy white blood cells
- Autoimmune disorders such as sarcoidosis, lupus, rheumatoid arthritis
- Nutrient deficiencies
The causes listed here are commonly associated with this symptom, but low WBCs are not enough to identify the underlying condition responsible. Work with your doctor or other health care professional for an accurate diagnosis.
Why Are High Levels of White Blood Cells Bad?
Your doctor will interpret your WBC test results, taking into account your medical history and other tests results. A result that is slightly high may not be of medical significance, as this test often varies from day to day and from person to person.
If you are worried about a high WBC count, work with your doctor to determine and address the underlying causes and develop an appropriate treatment plan.
White blood cells are immune cells capable of causing oxidative stress and damage.
Leukocytes participate in the inflammation process, are recruited at the site of endothelial injury, and form foam cells in the plaque .
Interleukins and tumor necrosis factor-α are released from activated leukocytes and can cause endothelial dysfunction .
White blood cells can build up in the arteries and cause hardening, contributing to plaques and potentially heart attacks. Besides hardening of the arteries, high levels of white blood cells can cause problems with circulation, blood thickening, and increased release of proinflammatory cytokines.
The following is a list of links to conditions or diseases that are exacerbated by high levels of white blood cells:
Repeatedly, studies have shown that WBC is a clinical marker of inflammation and a strong predictor of the risk of dying (mortality) from all causes [4, 5].
The total WBC count is an independent predictor of mortality in older adults, but the monocyte subtype provides greater predictability .
2) Heart Disease
WBC counts were almost linearly associated with cardiovascular mortality .
WBC counts within 24 hours of hospital admission for a heart attack is a strong and independent predictor of in-hospital and 30-day mortality risk. Relative to those patients in the lowest 20%, patients in the highest 20% of WBC counts were 3 times more likely to die at 30 days .
Circulating WBCs amplifies oxidative stress in heart failure .
A high WBC count was also significantly associated with cancer-related mortality .
A high WBC value predicted diabetes when adjusted for age, sex, body fat, and other established predictors of diabetes .
Also, a high WBC count at baseline was associated with a subsequent worsening of insulin resistance and the development of type 2 diabetes in Pima Indians .
These findings are consistent with the hypothesis that chronic activation of the immune system may play a role in causing type 2 diabetes .
An increased neutrophil count and reduced lymphocyte count are independent predictors of increased mortality risk in dialysis patients .
Factors that Increase White Blood Cell Levels
If you have low white blood cell levels, the first thing to do is to work with your doctor to address any underlying causes. You may try the additional strategies listed below if you and your doctor determine that they could be appropriate. None of these strategies should ever be done in place of what your doctor recommends or prescribes!
1) Consult Your Doctor
Low WBCs often indicate an underlying health problem that must be addressed, and your doctor is the best resource to figure out the appropriate treatment and management strategies. You and your doctor may discuss medications, supplements, or alternative treatments like acupuncture or sauna bathing, depending on what’s causing your WBCs to be low.
Meanwhile, if you think your immune system may be compromised, take extra precautions with your hygiene. Make sure you wash your hands and avoid contact with people who may be sick.
2) Heavy Resistance Exercise
During and after exercise in weight training males, WBC increased. This shows that leukocyte counts can significantly increase in response to heavy-resistance training/exercise .
During the recovery phase after submaximal exercise, there was an increase in the numbers of total leukocytes, neutrophils, lymphocytes, and monocytes .
Be cautious with exercise, particularly if your low WBC count is caused by an underlying illness. Your doctor can help you determine if your body can handle this kind of stress.
3) Healthy Diet
A healthy diet rich in fruits and vegetables can help balance your immune system and maintain optimal health. Fruits and vegetables contain important vitamins, minerals, and polyphenols.
Some foods and nutrients have been specifically found to increase WBC count in animal models. These include:
- Vitamin B12 helps to increase WBC counts. However, this effect was only seen in rats with protein deficiency; rats who ate a normal level of protein had unchanged WBC counts .
- Garlic, which slightly increased total WBC in rats .
- Noni juice, which increased WBC and other blood and platelets counts in rats .
- Fish oil, which increased WBCs in broiler chickens .
Folate deficiency is associated with decreases in lymphocytes and granulocytes, two types of white blood cells .
In a randomized clinical trial (RCT), folic acid supplementation increased WBC counts in children receiving the anti-seizure medication carbamazepine .
5) Shark Liver Oil
Shark liver oil has a lot of alkylglycerols, which are fats that stimulate the production of WBCs. Shark liver oil supplementation was shown to increase WBC counts in humans .
Substances and Pathways That Increase WBC Levels
These are the natural regulatory pathways that normally stimulate white blood cells in your body. While they are associated with increased WBCs, there is little evidence that attempting to modulate them will affect your immune system in any significant or beneficial way.
Talk to your doctor about the most appropriate strategies to treat underlying conditions and manage your white blood cell count.
1) Cortisol/GlucocorticoidsGlucocorticoids/cortisol can increase the number of WBCs in the body .
Glucocorticoids (includes cortisol) increase leukocytes/WBCs in blood but decrease the number of lymphocytes, eosinophils, monocytes, and basophils .
A single dose of cortisol resulted in a 70% decrease in lymphocytes and a 90% decrease in monocytes, occurring 4 to 6 hours after treatment and persisting for about 24 hours. Cell numbers then rise 24 to 72 hours after treatment .
We strongly recommend against using cortisol to increase WBCs. It is always preferable to talk to your doctor about strategies to treat underlying conditions causing abnormal WBCs.
2) LeptinWBCs are correlated to the amount of body fat in humans.
Leptin stimulates myeloid production, which is a precursor of WBCs .
Leptin is a hormone found in fat cells. The amount that circulates in humans is in direct proportion to the percentage of body fat. Leptin and the leptin receptor work together to stimulate blood cell production .
In Pima Indians, the WBC count is positively correlated with the percentage of body fat. The more fat the subjects had, the more WBCs they had in their bodies .
Weight loss can reduce the WBC count .
After controlling for age and gender, percent body fat accounted for 23% of the variance in the WBC count .
3) Adrenaline and Noradrenaline
Adrenaline and noradrenaline were infused into 5 healthy subjects. Total WBC counts increased in response to adrenaline and noradrenaline. Both alpha- and beta-adrenergic receptors are involved in the mobilization of lymphocytes .
4) MSHMSH is capable of increasing WBCs in diabetic rats, who had lower levels .
5) Thyroid Hormones
Thyroid hormones may slightly increase WBCs. People who are hypothyroid have slightly lower WBC counts and it increases when they normalize their levels .
6) IGF-1IGF-I stimulates a rapid increase in a number of immune cells, including WBCs, and helps increase the overall effectiveness of the immune system .
IGF-1 has an antiapoptotic (inhibits cell death) effect on WBC count and may increase their number .
7) Growth Hormone
Growth hormone, like IGF-I, also stimulates a rapid increase in a number of immune cells, including WBCs, and helps increase the overall effectiveness of the immune system .
Growth hormone alone (administered via injection to rats) increased WBC count. This was due to an increase in the number of lymphocytes and monocytes .
8) ProlactinProlactin has a variety of immune-stimulating effects .
Injection of prolactin in mice increases WBCs .
Prolactin goes up after people have a seizure – and so do WBCs (but the authors didn’t connect the two) .
9) InsulinThe WBC count is related to blood insulin levels and insulin resistance in healthy individuals of Pima Indian descent. Higher insulin levels correlated to higher WBC counts .
However, this could simply be because WBCs increase insulin resistance, and insulin levels are higher in insulin-resistant states .
Using liver and fat cells from mice and humans as well as live mouse models, scientists discovered that an enzyme secreted by neutrophils called neutrophil elastase (NE) impaired insulin signaling and boosted resistance. Conversely, deletion of NE in obese mice fed a high-fat diet improved insulin sensitivity .
Factors that Decrease White Blood Cell Levels
Work with your doctor to treat any underlying conditions causing unusual WBC levels. You may try the additional strategies listed below if you and your doctor determine that they could be appropriate. None of these strategies should ever be done in place of what your doctor recommends or prescribes!
1) Reduce Stress
Stress can increase cortisol, adrenaline, and prolactin, all of which can increase WBCs. Reducing stress can decrease these hormones and help normalize high WBC counts [35, 36].
2) Improve Sleep Quality
Interrupted or poor quality sleep can increase the markers of inflammation, including white blood cell count. Make sure to get enough sleep at around the same time each night to help keep inflammation down [37, 38].
3) Stop Smoking
WBCs in male smokers were significantly higher than in ex-smokers and non-smokers. In current cigarette smokers, WBCs were related to the number of cigarettes smoked, the amount of inhalation, and the duration of smoking .
Obviously, smoking is not recommended as a means to increase WBCs. If you smoke, we recommend talking to your doctor about strategies to quit.
4) Reduce Simple Carbs
High carb meals are capable of increasing leptin, insulin and IGF-1, more so than by consuming fat. The high-carb “Western diet” has also been associated with increased WBC count and increased inflammation. Reducing carbs in your diet may help bring down elevated WBCs, reduce inflammation, and possibly prevent insulin resistance [40, 41, 42, 43].
5) Reduce Calories
Restricting calories in the long term has been linked to lower WBC counts without harmful effects to the immune system. In a two-year study of 218 healthy adults, the calorie-restricted adults, those who restricted calories also experienced a 0.6 unit reduction in WBCs. The effect of caloric restriction on people with elevated initial WBC counts is unknown .
Substances and Pathways
These are the natural substances and pathways that normally regulate white blood cells in your body. While they are associated with increased WBCs, there is little evidence that attempting to modulate them will affect your immune system in any significant or beneficial way.
Talk to your doctor about the most appropriate strategies to treat underlying conditions and manage your white blood cell count. None of these strategies should be used to replace your doctor’s recommendations!
Zinc plays an important role in immune function. Zinc deficiency increased the number of total WBCs, granulocytes (neutrophil, eosinophil, and basophil), and monocytes in mice without changing the number of lymphocytes .
Zinc deficiency induces the overall stress response which may result in increased WBCs .
Therefore, supplementation with zinc (if you’re deficient) may help decrease high WBCs.
WBCs decreased in men who ate a high-selenium diet. The decrease may have been due to changes in granulocytes .
Optimal Reference Range for White Blood Cells
Lab results are commonly shown as a set of values known as a reference range, which is sometimes referred to as a “normal range.” A reference range includes upper and lower limits of a lab test based on a group of otherwise healthy people.
Your healthcare provider will compare your lab test results with reference values to see if any of your results fall outside the range of expected values. By doing so, you and your healthcare provider can gain clues to help identify possible conditions or diseases.
Participants in a prospective cohort study with baseline WBC < 3,500 and WBCs > 6,000 had higher mortality than those in the 3,500 to 6,000 (WBC/mm^3) range .
In an additional independent study that covered 7 countries, results concluded that each point increase in WBCs was associated with 21% higher 5-year heart disease mortality (after adjustment for risk factors) .
A WBC count between 3.5 (3,500) and 6.0 (6,000) is considered normal.
WBC counts of 9,000 to 10,000 showed a 3.2 fold increase for the risk of cardiovascular disease mortality compared to those with WBC counts of 4,000 to 4,900 .
WBCs can vary from person to person based on a multitude of factors.
Irregular WBC Levels?
LabTestAnalyzer helps you make sense of your lab results. It informs you which labs are not in the optimal range and gives you guidance about how to get them to optimal. It also allows you to track your labs over time. No need to do thousands of hours of research on what to make of your lab tests.
LabTestAnalyzer is a sister company of SelfHacked. The proceeds from your purchase of this product are reinvested into our research and development, in order to serve you better. Thank you for your support.
'Radiation can cause cancer in most parts of the body, in all animals, and at any age, although radiation-induced solid tumors usually take 10–15 years, and can take up to 40 years, to become clinically manifest, and radiation-induced leukemias typically require 2–9 years to appear'.
DOPAMINE IS BIG IN ALL KINDS OF DISEASE VECTOR STUDIES ESPECIALLY BEHAVIOR LIKE DRUG ADDICTION OR FOOD/ALCOHOL ADDICTION.
'In addition to the classic thyroid symptoms that an individual was already experiencing, brain and neurotransmitter symptoms typically associate with either the neurotransmitter serotonin, or the neurotransmitter dopamine'.
The FEEDBACK from NOSY NEIGHBORS AND THE GANG since JAN 2019 HITTING creating PSYCHO-SEXUAL TORTURE came with lots of trying to MAKE ME MAD----MAKE ME ANGRY-----MAKE ME VIOLENT-----inability to sleep---unable to have friends/anti-social=====feeling of worthlessness-----low sex drive-----suicidal thoughts
MAN-----FEEDBACK MADE ME SOUND LIKE A MESS!
ACTUALLY, I AM THE SAME WELL-BALANCED---SELF-ACTUALIZED PERSON I WAS BEFORE ALL THESE IMPLANT MANIPULATIONS.
The THYROID is being attacked by these BRAIN IMPLANTS and it creates all kinds of SYMPTOMS----especially those SHE LOOKS CRAZY symptoms.
- 'Lachrymal glands: In eye secrete water to moisten the eye'.
It's those GLAZED EYES with DILATED PUPILS!
Hypothyroidism leads to a decrease in thyroid hormone production.
The tissues of the brain are saturated with thyroid hormone receptor sites, or places that accept thyroid hormones in order to fulfill physiological mechanisms. When the brain does not receive thyroid hormones into the receptor sites, neurotransmitter production declines, leading to additional symptoms related to brain chemistry imbalance. This imbalance perpetuates an inflammatory cascade which leads to further brain degeneration, fewer neurotransmitters, and further inflammation. This of course leads to more symptoms.
In addition to the classic thyroid symptoms that an individual was already experiencing, brain and neurotransmitter symptoms typically associate with either the neurotransmitter serotonin, or the neurotransmitter dopamine.
Serotonin deficiency symptoms can include:
Increased susceptibility to pain
Lack of enthusiasm to favorite foods, friends, activities, etc.
Feelings of being overwhelmed
Inability to fall into a deep/restful sleep
Feelings of dependency on others
Difficulty enjoying relationships
Dopamine deficiency symptoms can include:
Feelings of worthlessness and hopelessness
Inability to handle stress
Decreased sex drive
Short tempered/lose temper for minor issues
Desire to isolate one’s self from others
Lack of concern for loved ones
Self destructive thoughts
Increased aggression, especially under stress
Need for caffeine to be alert
15 Types of Glands in Human Body & their Functions
Glands are studied as a part of human anatomy and physiology.
They are sac-like tissue structures in the body which play a vital role in the physiology and homeostasis.
Anatomically they are made of secretory cells and also situated in a safe yet prominent location inside the body.
Glands are quite numerous and regulate different physiological functions and activities.
The glands can be differentiated based on their locations, based on their type of secretions, based on the organ systems they control, etc.
These glands mostly perform their function through the release of secretions. These secretions can be hormones, enzymes and sometimes just fluid yet all of them have some essential functions.
Without their secretions, there can arise enzyme and hormone deficiency diseases.
1. Exocrine glands: They secrete into a location or region of the body through a duct, and their secretions are called enzymes mostly while some are non-enzymes.
Types of exocrine glands and their secretions include
- Salivary gland: In the buccal cavity secrete saliva. This saliva has many functions.
- Pancreas: Secretes enzyme amylase, trypsin, and lipase. These enzymes digest carbohydrates, proteins, and fats respectively.
- Sperm: Sperms produce Hyaluronidase enzyme by which they swim in the uterine tissue to reach ova. The other application of this enzyme is to help the drug reach inner most tissues of the body.
- Sweat glands: secrete sweat which acts to regulate body temperature and also excretion.
- Sebaceous gland: secrete sebum in the skin and the ear.
- Lachrymal glands: In eye secrete water to moisten the eye.
They are classified under an endocrine system of the 12 body systems.
Based on their location there are broadly two types of endocrine glands viz.
a) Pituitary Gland (of the hypothalamus): This is a gland located below the hypothalamus in the brain.
For more about anatomy refer to the pituitary structure.
The gland is differentiated as anterior and posterior gland and has definite secretions for each type i.e.
i) Anterior pituitary gland: Located in the front part of the pituitary
- Prolactin: Stimulates milk production in mothers.
- Somatotrophin: A hormone which regulates the growth of the body and tissues.
- Luteinizing hormone: Stimulates ovulation (egg formation) in females and testosterone production in males.
- Thyroid stimulating hormone: Stimulates thyroid gland to produce T3 & T4 hormones.
- Adrenocorticotrophic hormone: Stimulates secretion of glucocorticoids and mineralocorticoid hormones.
- Follicle stimulating hormone signals formation of estrogen and progesterone by the female reproductive system.
ii. Posterior pituitary
Present behind the front part of pituitary as shown in the diagram above.
- Vasopressin (anti-diuretic hormone) It controls water loss from kidneys. So minimizes urine formation and saves water in the body.
- Oxytocin It signals uterus for delivery in pregnant women after the period of gestation. It also stimulates milk secretion in mother.
b) Thyroid gland: Produces thyroid hormones T3 & T4.
c) Parathyroid gland: Produces parathormone.
Both thyroid and parathyroid glands are examples for types of glands in the neck.
d) Adrenal gland: located on the two kidneys. It has an outer cortex and inner medulla regions.
- Cortex: secretes glucocorticoids and mineralocorticoids.
- Medulla: secretes nor-adrenalin. This is one of the neurotransmitters and is called a flight or fight hormone.
f) Kidneys: produce Renin-angiotensin (controls blood pressure).
g) Pineal gland: Located inside the brain and called as natural body clock. One can see jet-lag effects due to its secretions. Melatonin (sleep hormone).
You may also like………> Hormones in the human body.
h) Testes: Secrete Testosterone. Produced only in males to a large extent and to a minimum extent in women. It signals male characters like the beard, heavy muscles, etc.
i) ovaries: Produce Estrogen, progesterone. Secreted only in women and regulate reproduction cycle.
Reference: Principles of Anatomy and Physiology by Gerard J. Tortora
All the glands are located in distinct locations in the human body. They regulate the physiology to a large extent by receiving stimulus from the brain. So we can notice that those glands present in the brain secrete hormones which go outside the brain and act on other glands distributed in different parts of the body. They travel through the blood to the target organs after being released.
I tell my GYN ----I don't care if I am LOW LIBIDO ----I like my natural state----and what do barber surgeons do? They dose me with IMPLANT MANIPULATION of GLANDS secreting TESTOSTERONE.
So, my voice deepens ----my chin hair grows---- my underarm sweat glands GIVE A MANLY STINK. Heavy sweating with strong manly odor hit me back in 2010---2015.
Fast forward today----chin hair does get worse-----underarm sweat and stink goes away----and I am back to normal.
Think about MEN losing facial or body hair----women growing facial hair body hair and global banking 1% BARBER SURGEONS say---
WE HAVE A PATENTED PRODUCT---MEDICAL PROCEDURE JUST FOR YOU!
SHE ACTS SO ODD------LOOK, SHE IS CRAZY.
Well, people who are creative are being attacked by HOSTING SERVER NOSY NEIGHBORS AND THE GANG!
Heaven forbid---she is THINKING.
John Lennon on Being a Genius
"If there's such a thing as genius...I am one!" Taken from the 1971 Rolling…'